Hyperkaliämie: Ätiologie von Amy Sussman, MD

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Über den Vortrag

Der Vortrag „Hyperkaliämie: Ätiologie“ von Amy Sussman, MD ist Bestandteil des Kurses „Kaliumstörungen: Hypo- und Hyperkaliämie“.


Quiz zum Vortrag

  1. Increased osmolarity
  2. Metabolic acidosis
  3. Tissue breakdown
  4. Decreased activity of Na/K ATPase
  5. Increased activity of K/H ATPase
  1. Digitalis
  2. Metabolic alkalosis
  3. Insulin
  4. Hyposmolarity
  1. Aldosterone blockade increases the risk of hyperkalemia in patients with oliguric renal failure.
  2. Heart failure will cause volume overload, which will decrease serum potassium levels.
  3. Ifosfamide is associated with hyperkalemic renal tubular acidosis.
  4. Urinary tract obstruction impairs the outer medullary potassium channels in the principal cells, which will increase potassium excretion.
  1. Sickle cell disease - functional hypoaldosteronism
  2. Amiloride - direct renin inhibitor
  3. Tumor lysis syndrome - pseudohyperkalemia
  4. Renal tubular acidosis type 1 - hyperkalemia
  5. Hyperkalemic periodic paralysis - dihydropyridine calcium channels

Dozent des Vortrages Hyperkaliämie: Ätiologie

 Amy Sussman, MD

Amy Sussman, MD

Dr. Amy Sussman is an Associate Professor of Medicine and the Vice Chair of Education in the Department of Medicine of the University of Arizona (UA), USA.
She obtained her MD from the University of Arizona in 2001 and completed residency and a Nephrology Fellowship at the University of Washington. Dr. Sussman was recruited back to the UA in 2009 and also serves as her department's clerkship director, and as the program director of the UA Nephrology Fellowship.
Because of her strong emphasis on medical student teaching, she has been distinguished with multiple teaching awards in the UA College of Medicine.
Within Lecturio, Dr. Sussman teaches courses on Nephrology.


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